The Japanese journal of neuropsychology

Full Text of this Article
in Japanese PDF (364K)

ArticleTitle	Transcortical sensory aphasia caused by occlusion of the posterior cerebral artery
Language	J
AuthorList	Yuichiro Inatomi¹⁾, Toshiro Yonehara¹⁾, Shodo Fujioka¹⁾, Makoto Uchino²⁾ Koichi Tagawa³⁾
Affiliation	1) Stroke Center, Saiseikai Kumamoto Hospital 2) Department of Neurology, Kumamoto University School of Medicine 3) Fukuoka Higher Brain Function Center, Nagao Hospital
Publication	Japanese Journal of Neuropsychology: 17 (1), 76-81, 2001
Received	Feb 14, 2000
Accepted	Oct 19, 2000
Abstract	We presented two cases with transcortical sensory aphasia associated with the occlusion of the left posterior cerebral artery (PCA). In patient 1, the size of the critical lesion was larger than the whole PCA territory, including the left temporo-parieto-occipital junction due to poor collateral circulation from the left middle cerebral artery (MCA). In patient 2, cerebral angiogram on admission showed the occlusion of the left PCA and the left internal carotid artery. Single photon emission computed tomography on day 7 revealed the reduction of blood flow in the left temporal lobe and the temporo-parieto-occipital junction. Magnetic resonance imaging on day 12, however, demonstrated the infarct lesion limited in the left medial temporal lobe, when his aphasia improved. It was suggested that the reperfusion in the temporo-parieto-occipital junction was accounted for the improvement of his aphasia. In conclusion, the lesions responsible for the transcortical sensory aphasia associated with the PCA occlusion requires being large in size, and including left temporo-parieto-occipital junction, which is the watershed area between the MCA and PCA.
Keywords	transcortical sensory aphasia, posterior cerebral artery, borderzone infarction